Rickets Sentence Examples

Rickets, scurvy and "marasmus" may be instanced as diet diseases in children.

Owing to this influence it has been used in rickets and osteomalacia.

The most important disease hitherto undescribed was rickets, first made known by Arnold de Boot, a Frisian who practised in Ireland, in 1649, and afterwards more fully in the celebrated work of Francis Glisson (1597-1677) in 1651.

Rickets results from a lack of vitamin D rich foods in a child's diet, insufficient sunlight exposure, or digestive system problems that result in poor vitamin D absorption.

Thus the liability to tubercular infection is far commoner in the midst of a depraved population than in one fulfilling the primary laws of nature; rickets is a disease of great cities rather than of rural districts; and syphilis is more disastrous and protracted in its course in the depraved in health than in the robust.

This is because Vitamin D, a vitamin which prevents rickets, is partly made in skin.

A deficiency of Vitamin D leads to a failure of the bones to grow and causes rickets in children and osteoporosis in adults.

They tend to overprotect their children, with much greater danger to their personal and emotional development than the risk of developing rickets.

Also, the hundreds of children in our community never had rickets either.

He published at Oxford in 1668 two tracts, on respiration and rickets, and in 1674 these were reprinted, the former in an enlarged and corrected form, with three others "De sal-nitro et spiritu nitro - aereo," "De respiratione foetus in utero et ovo," and "De motu musculari et spiritibus animalibus" as Tractatus quinque medico-physici.

Vitamin D deficiency causes rickets, a disease of the bones.

Vitamin D deficiency can cause rickets, which can lead to permanently stunted or irregular growth.

Malnourished infants and children may develop rickets, a severe vitamin D deficiency that results in bone pain, malformed bones, and poor dental health.

It is characterized by softening of the bone and is sometimes referred to as adult rickets.

Lack of calcium can cause rickets, a condition in which the bones are soft and develop in abnormal shapes.

When vitamin D deficiency continues for many months in growing children, the disease commonly referred to as rickets occurs.

If the deficiency occurs for a period of many months or years, however, rickets may develop.

The symptoms of rickets include bowed legs and bowed arms.

Other symptoms of rickets include bony bumps on the ribs called rachitic rosary (beadlike prominences at the junction of the ribs with their cartilages) and knock-knees.

Seizures may also occasionally occur in a child with rickets, because of reduced levels of dissolved calcium in the bloodstream.

The doctor should be called if the parent notices that the child has any signs of vitamin D deficiency or rickets.

Rickets is diagnosed by x-ray examination of the leg bones.

A distinct pattern of irregularities, abnormalities, and a coarse appearance can be clearly seen if a child has rickets.

Rickets heals promptly with large doses vitamin D administered orally each day for approximately one month.

Rickets are usually treated with oral supplements of vitamin D, with the recommendation to acquire daily exposure to direct sunlight.

Rickets may also occur with calcium deficiency, even when a child is regularly exposed to sunshine.

This type of rickets has been found in various parts of Africa.

The bone deformities are similar to, or are the same as, those that occur in typical rickets; however, calcium deficiency rickets is treated by increasing the amount of calcium in the diet.

No amount of vitamin D can cure the rickets of a child with a diet that is extremely low in calcium.

The prognosis for correcting vitamin D deficiency and rickets is excellent.

It is often seen in children with rickets.

It is essential for a number of processes in your body, as well as for the prevention of rickets.

Inadequate vitamin D can lead to a number of health problems besides rickets, including increased risk of coronary disease and certain cancers.

Rickets is a severe bone disorder causes by persistent vitamin D deficiency.

This is why one of the classic diseases of vitamin D deficiency is rickets.

A deficiency in vitamin D can result in rickets.

Children who receive too little vitamin D are at risk for developing rickets, a devastating condition which can cause bones to weaken and break.

Deficiencies in children can result in the bone-weakening disease rickets.

Children with rickets often cry because their bones ache and hurt, but adults may be walking around with insufficient amounts of vitamin D and not feel a thing.

The most severe form of vitamin D deficiency is rickets, a softening and weakening of the bones that occurs in children.

Rickets can lead to mild to severe bone deformities.

Not enough vitamin D in the body can cause thin and brittle bones, which leads to rickets in children and a very common disease in adults called osteoporosis.

Asked about Rickets 28, What causes of leg length discrepancy do you know?

Vitamin D Babies who do not get enough vitamin D may get rickets, a disease that affects bone development.

The disease caused by a low calcium diet is called rickets.

Females who suffered from a distorted pelvis as a result of childhood rickets often experienced very difficult and sometimes fatal births.

A vitamin D deficiency can lead to osteoporosis in adults or rickets in children.

Specific diseases causing an increased risk for fractures include Paget's disease, rickets, osteogenesis imperfecta, osteoporosis, bone cancer and tumors, and prolonged disuse of a nonfunctional body part such as after a stroke.

In-born errors of vitamin D metabolism can also cause vitamin D deficiency and rickets; these children cannot convert inactive vitamin D to active vitamin D and suffer the same symptoms as children with a nutritional deficiency.

It is for this reason one of the symptoms of [ins.lovetoknow.com/What_Causes_Vitamin_D_Deficiency vitamin D deficiency] is rickets, which is a softening of the bones.

Similarly Karl Hoffmann of Wiirzburg wasted his appreciations of the newer schools of developmental biology in fanciful notions of human diseases as reversions to normal stages of lower animals; scrofula being for him a reversion to the insect, rickets to the mollusc, epilepsy to the oscillaria, and so forth.