Most Labor MPs, including the whole front bench, then abstained on the substantive vote.
The MPS would seek clarification on the admissibility of previous unproven allegations of misconduct.
Only the Tories and a tiny minority of the labor MPs would support a war without explicit UN authorization.
The MPs voted by 317 to 145 for an outright ban.
Field sport campaigners claim Labor MPs pressing for a total hunting ban are acting out of class hatred rather than concern for animals.
bloody DOCKING NONE of our MPs would deliberately smash a dog's tail and leave it a bloodied mess.
Mr Blair told MPs in the House he was not playing a game of " dangerous brinkmanship " with President Saddam.
Some MPS Pentium systems, and all MPS Pentium Pro and Pentium II systems, have independent L2 caches.
On Wednesday 29 October, MPs voted by 316 to 160 in support of reclassifying cannabis.
chloride of lime were hung at the windows of the House of Commons to prevent MPs getting cholera.
Sheets soaked with chloride of lime were hung at the windows of the House of Commons to prevent MPs getting cholera.
I warned Tory MPs not to get too cocky about the police investigations.
Were MPs and cabinet members really comfortable with the pager system enforcing a uniformed linguistic and ideological conformity from them?
conservationists today warned MPs not to fall into a TB trap set by farming unions.
Will the speaker sometimes exclude all non-English constituency MPs, and other times only the Scottish, or perhaps only the Welsh and Scottish?
criticiseparliamentary tactics were openly criticized by some of his own MPs.
I also experienced my first lobbying meeting with a Government Minister, as part of a cross-party delegation of Scottish MPs.
deluge of rote letters from MPs adds to the burden.
desultory discussion in 1994, a committee of 14 MPs nodded through the Units of Measurement Regulations.
Too long, it turns out, for the 49 Labor MPs who defeated the government on its plans for 90-day detention.
eleventh in a table of Scottish MPs activities, with 298 contributions in the past year.
Will the newly emboldened Labor MPs:: pursue this?
famed as a place for the meeting of minds for hacks and MPs.
Any positive lessons to be learned will be fed into appropriate fora in the MPS for further detailed study.
The harsh fact is that both MPs find it easier to blame foreigners than motivate at home.
foreshortened view makes MPs look like pygmies shouting from the bottom of an untidy green pit.
A conservative frontbencher said: " Labor MPs are furious.
MPs qualify for a fantastically generous final salary pension scheme, with benefits underwritten by the public purse, of course.
The group consists of at least 25 like-minded Conservative MPs and it has recently grilled Tory leadership hopefuls about their core beliefs.
MPs in Leeds and elsewhere would be very foolish to ignore enormous groundswell of opinion against the war.
Not only is there this huge groundswell of support from Mercury readers, we have cross-party support from local MPs.
heckles the candidates In half the country, sitting Labor MPs will defend their seats at the next election.
Tuesday 19 October 1971 A group of five Northern Ireland Members of Parliament (MPs) began a 48 hour hunger strike against Internment.
inadequacy MPS review failed to expose these inadequacies.
More than 100 MPs have called for an independent inquiry into the scandal.
interdenominational human rights pressure group which has been supported by more than 100 MPs.
MPs, understandably, were less enthusiastic about having nosey interlopers in their midst.
landslide majority of 167 MPs.
leaked memo last night, causing anger among MPs.
He will most likely take steps to keep his party's MPs on a shorter leash from now on.
I hear that most MPs refused even to meet these lobbyists.
localism made by a group of our new MPs and candidates.
Details of the schemes emerged in a leaked memo last night, causing anger among MPs.
misreported by the media as the union giving money to the new group of MPs.
I bought a hydraulic cable oiler from MPS for 10.99.
Winston Churchill led a number of MPs who bitterly opposed it.
The English MPs can always outvote the Scots on anything in Westminster.
A revolt by Labor MPs has inflicted the first ever parliamentary defeat on the government on its anti-terror laws.
police officer recruitment targets for 2003/2004 7. The MPS needs to recruit a total of 2,840 officers during 2003/2004.
Price £ 21 2) Navy blue polo shirt with embroidered MPS logo on the front.
proportional representation nor the right to recall MPs.
CPS lawyers at the point of charge 8. All MPS Boroughs now have access to a duty prosecutor 9-5 Monday to Friday.
Well done to MPS member Caroline Hartley for winning the raffle.
The legislation establishing NHS foundation trusts attracted much controversy, and a major backbench rebellion among Labor MPs, but was passed into law.
With 3 MPS members (Damian, Franco and Dave) going to see a former MPS member I recon it does.
MPs must remain resolute in resisting any such extension.
The Planning process was very challenging and there was a major restructure of the MPS planning team involved part way through the year.
The all-woman shortlist, in short, has been a crucial weapon in the fight to secure more women MPs representing Welsh seats.
Many on the loyal left were quietly hoping that the government would use a sleight of hand to deny MPs the chance to protest.
These two affluent cities, which regularly used to send Conservative MPs to Westminster, again spurned the Tories at May's local elections.
The video was primarily produced for, and distributed to, all DFID staff, external stakeholders and MPs.
Mr Blair yesterday faced down rebellious Labor MPs with a passionate defense of his hardline stance on Iraq.
I think we should ask the female MPs in parliament today if they have forgotten the suffragettes.
Even the normally supine New Labor MPs in Leeds have complained to the Government.
supine nature of MPs.
up-and-coming talent is a special priority at the MPS.
At the time, MPs thought the issue revolved solely around RUC officers.
EXCLUSIVE MPs slam rip-off charges for patients Sunday Mirror, UK - 15 Jul 2006 By Vincent Moss And Stephen Hayward.
This presents the MPS with a major opportunity to prevent money being siphoned illegally from the public services and the capital 's economy.
These two affluent cities, which regularly used to send Conservative MPs to Westminster, again spurned the Tories at May 's local elections.
In the end it was the MPs who got us our stay of execution.
The biggest threat to British democracy is actually not the EU, but the supine nature of MPs.
All that is needed is for MPs to say: Pause for a moment, let us have a proper, truthful explanation.
The promotion of up-and-coming talent is a special priority at the MPS.
A majority of Tory MPs will vote in favor, he believes.
Minimum Performance Standards (MPS) are evaluated following every Summer National Senior Games to establish a method whereby an athlete can qualify for the Summer Games by meeting or exceeding the standard.
In general, the MPS is based on the time, distance, or score of the 6th place finisher, plus or minus 10 percent.
You'll find some interesting equipment on the MPs, including adult diapers and condoms.
Mucopolysaccharidosis (MPS) is a general term for many different related inherited disorders that are caused by the accumulation of mucopolysaccharides in body tissues.
There are many different types of GAGs, and different GAGs are unable to be broken down in each of the MPS conditions.
Before specific deficient enzymes were identified, MPS disorders were diagnosed by the signs and symptoms seen in an individual.
The discovery of individual enzyme deficits resulted in a reclassification of some of the MPS disorders.
Types of MPS disorders are MPS I, MPS II, MPS III, MPS IV, MPS VI, MPS VII, and MPS IX.
MPS disorders are rare, and the frequency with which they occur varies depending on the type of the disorder.
For all MPS types combined, the disorder occurs in only about one of every 25,000 people.
Except for MPS II, individuals of both genders are affected equally.
Because of its inheritance pattern, MPS II is found only in males.
All MPS disorders are present at birth, although symptoms appear at different times, depending on the type of disorder.
There appears to be no race or ethnic component in the distribution of MPS disorders.
All MPS disorder except MPS II are inherited in an autosomal recessive manner.
If the parent has one good copy of the gene and one defective copy, the parent will not have MPS and may be unaware that he or she has a defective gene.
MPS only occurs when both of an individual's genes that produce the same enzyme contain a mutation or defect, causing them not to function properly.
Some individuals who have MPS are able to have children.
Children of MPS parents are all carriers of the disorder, because they inherit one bad copy of the gene from the affected parent.
Unlike the other MPS conditions, MPS II is inherited in an X-linked recessive manner, which means that the gene causing the condition is located on the X chromosome, one of the two sex chromosomes.
Although MPS are all inherited disorders, each type is caused by a deficiency of one particular enzyme involved in breaking down GAGs.
The accumulation of the GAGs in the tissues and organs in the body causes the symptoms characteristic of the MPS disorders.
MPS I is caused by a deficiency of the enzyme alpha-L-iduronidase.
This tends to be the most severe form of MPS I.
Hurler-Scheie syndrome an intermediate form of MPS I, meaning that the symptoms are not as severe as those in individuals who have MPS I H but not as mild as those in MPS I S.
They can have normal intelligence; however, some individuals with MPS I H/S experience learning difficulties.
The prognosis for children with MPS I H/S is variable with some individuals dying during childhood, while others live to adulthood.
Scheie syndrome is the mild form of MPS I.
Individuals with MPS I S usually have normal intelligence, although there have been some reports of individuals with MPS I S developing psychiatric problems.
Individuals with MPS I S do not develop the facial features seen with MPS I H and usually these individuals have a normal life span.
Like many MPS conditions, Hunter syndrome is divided into two forms, mild and severe.
The severe form of MPS II is associated with progressive mental retardation and physical disability, with most individuals dying before age 15.
These symptoms progress at different rates depending on whether the individual has the mild or severe form of MPS II.
MPS III, like the other MPS conditions, was initially diagnosed by the individual having certain physical signs and symptoms.
MPS III is in the early 2000s subdivided into four groups, labeled A through D, based on the specific enzyme that is deficient.
MPS III is a variable condition, with symptoms beginning to appear between two and six years of age.
In MPS III, signs that the CNS is degenerating usually become evident between six and ten years of age.
Many children with MPS III develop seizures, sleeplessness, thicker skin, joint contractures, enlarged tongues, cardiomyopathy, behavior problems, and mental retardation.
The life expectancy in MPS III is also variable.
On average, individuals with MPS III live until they are teenagers, with some living longer and others not that long.
MPS IIIA (Sanfilippo syndrome type A) is caused by a deficiency of the enzyme heparan N-sulfatase.
Type IIIA is the most severe of the four types of MPS III.
A study in British Columbia estimated that one in every 325,000 babies is born with MPS IIIA.
MPS IIIA is the most common of the four types in Northwestern Europe.
The gene that causes MPS IIIA is located on the long arm of chromosome 17.
MPS IIIB( Sanfilippo syndrome type B) is due to a deficiency in N-acetyl-alpha-D-glucosaminidase (NAG).
This type of MPS III is not as severe as type IIIA, and the characteristic signs and symptoms vary.
The gene associated with MPS IIIB is also located on the long arm of chromosome 17.
MPS IIIC (Sanfilippo syndrome type C) is caused by a deficiency in the enzyme acetyl-CoA-alpha-glucosaminide acetyltransferase.
The gene involved in MPS IIIC is believed to be located on chromosome 14.
MPS IIID (Sanfilippo syndrome type D) is caused by a deficiency in the enzyme N-acetylglucosamine-6-sulfatase.
The gene involved in MPS IIID is located on the long arm of chromosome 12.
MPS IV A is the severe form of the disorder and is caused by a deficiency in the enzyme galactosamine-6-sulphatase.
The gene involved with MPS IV A is located on the long arm of chromosome 16.
The major organs affected by MPS IV are the cornea and the cartilage, particularly the cartilage of the neck.
Individuals with MPS IV appear healthy at birth but show skeletal deformities and growth retardation by age three.
MPS IV B (Morquio syndrome type B) is the milder form of the disorder.
The enzyme, beta-galactosidase, is deficient in MPS IV B.
Individuals with the MPS IV B can have normal lifespans (into their 70s).
MPS VI, which is another rare form of MPS, is caused by a deficiency of the enzyme N-acetylglucosamine-4-sulphatase.
Typically, the nervous system or intelligence of an individual with MPS VI is not affected.
Individuals with a more severe form of MPS VI can have airway obstruction, develop hydrocephalus (accumulation of fluid in the brain), and exhibit bone changes.
Individuals with a severe form of MPS VI are more likely to die while in their teens.
The gene involved in MPS VI is believed to be located on the long arm of chromosome 5.
MPS VII is an extremely rare form of MPS and is caused by a deficiency of the enzyme beta-glucuronidase.
The gene that causes MPS VII is located on the long arm of chromosome 7.
MPS IX, a condition first described in 1996, is caused by a deficiency of the enzyme hyaluronidase.
The gene involved in MPS IX is believed to be located on the short arm of chromosome 3.
Parents should inform the doctor immediately if MPS runs in their family, so that early testing can be done on their children.
While a diagnosis for each type of MPS can be made based on the physical signs described above, several of the conditions have similar features.
Therefore, enzyme analysis is used to determine the specific MPS disorder.
Enzyme analysis often cannot accurately determine if an individual is a carrier for an MPS disorder, because the enzyme levels in individuals who are not carriers overlaps the enzyme levels seen in those individuals who are carrier for MPS.
With many of the MPS conditions, several mutations have been found in each gene involved that can cause symptoms of each condition.
Once a couple has had a child with MPS, prenatal testing is available to them to help determine if another fetus is affected with the same MPS as their previous other child.
As of 2004 there was no cure for MPS, although several types of experimental therapies are being investigated in the early 2000s.
For MPS I and VI, bone marrow transplantation has been attempted as a treatment option.
For those types of MPS, bone marrow transplantation has sometimes helped slow down the progression or reverse some of symptoms of the disorder in some children.
However, bone marrow transplantation is not thought to be helpful in other MPS disorders.
Availability of donors is limited, and as a result, very few bone marrow transplantations are done for MPS.
Another experimental treatment for MPS I involves extended treatment with recombinant human alpha-L-iduronidase.
The course of this disorder varies with the specific type of MPS the individual has.
MPS I H is often fatal in childhood, with individuals rarely living past age 12.
Individuals with MPS I H/S may die in childhood or live to adulthood.
Individuals with MPS I H have health problems but usually have a normal lifespan.
Individuals with mild MPS II live relatively normal lives, while individuals with the severe form of the disorder usually die in their teens.
The life expectancy in MPS III and MPS IV is also variable, depending on the severity of the disorder.
Individuals with MPS VI often have shorter than average life spans.
As of 2004 MPS IX had been diagnosed so recently that little information is available.
No specific measures can prevent the gene mutations that cause MPS.
For some of the MPS diseases, biochemical tests may be able to identify healthy individuals who are carriers of the defective gene, allowing them to make informed reproductive decisions.
Prenatal testing can also diagnose MPS in the fetus, but this testing is normally done only when there is some reason to expect to find the disorder (e.g. family history of the disease).
Many individuals with an MPS condition have problems with airway constriction.
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