Immunoglobulin Sentence Examples

Immunoglobulin A (IgA) is activated early in response to invasion by bacteria and viruses.

This antibody, also called an immunoglobulin, may damage the nerve fibers.

All received intravenous immunoglobulin, and four received platelet concentrate.

However, there may be some protection even if you are given immunoglobulin up to 10 days after contact with the virus.

The use of Anti-D immunoglobulin has been one of the success stories of the last 20 years.

This is achieved by treatment with intravenous tetanus immunoglobulin.

The Immunization Division provides advice and supplies human normal immunoglobulin for contacts of cases.

In all patients receiving replacement immunoglobulin therapy, blood samples should be obtained at regular intervals to assess liver function.

In the case of severe exposure vaccination is often accompanied by injection of rabies immunoglobulin (IG ).

A randomized trial of intravenous immunoglobulin in inflammatory demyelinating optic neuritis.

Immunoglobulin JH gene rearrangement was detected in these lymphoma cells.

A temporary decline in T-cell subpopulations, but no reduction in serum immunoglobulin levels, could be observed.

Rapid evolution of immunoglobulin superfamily C2 domains expressed in immune system cells.

Properties of antibodies 1. Antibody structure; variable and constant domains; isotypes; immunoglobulin gene superfamily.

Passive immunization should be carried out using Varicella zoster immunoglobulin (VZIG) in non-immune patients if exposed to chickenpox or shingles.

Although 90 percent of those with ITP respond to immunoglobulin treatment, it is an expensive treatment.

Mast cells produce a special class of antibody, immunoglobulin E (IgE), that coats cell surfaces.

Immunoglobulin G (IgG)-Immunoglobulin type gamma, the most common type found in the blood and tissue fluids.

It displays a type of antibody called immunoglobulin type E (IgE) on its cell surface and participates in the allergic response by releasing histamine from intracellular granules.

A characteristic finding in children diagnosed with HSP is higher levels of immunoglobulin A (IgA) in the blood and deposits of IgA on the walls of the child's blood vessels.

It is the most common type of immunoglobulin in newborns and, therefore, the most useful indicator of the presence of one of the TORCH virus infections.

It sends out immunoglobulin E (IgE), an antibody, to destroy the eggs and protect the body, releasing histamines.

Decreased immunoglobulin levels (Iga, IgG, IgM).

The drawback of this test is that immunoglobulin levels are not always low for A-T, and they are also low in other conditions.

When the body is infected with the rubella virus, it produces both immunoglobulin G (IgG) and immunoglobulin M (IgM) antibodies to fight the infection.

This is done with injections of human tetanus immunoglobulin (TIG).

Specifically, an immunoglobulin antibody protein, IgE, is produced in response to the presence of the allergen.

Immunoglobulin E (IgE)-A type of protein in blood plasma that acts as an antibody to activate allergic reactions.

The most common tests performed to screen for hyper-IgM syndrome are a complete blood count (CBC) and a quantitative immunoglobulin test.

Immunoglobulin deficiencies refer to missing or reduced levels of immunoglobulin (IgG, IgA, IgM) associated with an inability to make adequate specific antibody.

Immunoglobulin G (IgG) is the most abundant class of immunoglobulins, directed toward viruses, bacterial organisms, and toxins.

Immunoglobulin M (IgM) is the first antibody produced in an immune response to any invading organism or toxic substance.

Immunoglobulin E (IgE) is found in respiratory secretions and is directed toward invasion of the body by parasites and in allergic reactions such as hay fever, atopic dermatitis, and allergic asthma.

Any disease that harms the development or function of B cells will, therefore, affect the production of immunoglobulin antibodies.

About 70 percent of immunoglobulin deficiencies involve B lymphocytes and 20-30 percent involve T lymphocytes.

Many of the infections that occur in children with immunoglobulin deficiency syndromes are caused by bacterial organisms or microbes.

In a healthy body with an adequately functioning immune system, immunoglobulin antibodies bind to the capsule and overcome the bacteria's defenses.

Immunoglobulin deficiency syndromes are primary immunodeficiency diseases.

All classes of immunoglobulin antibodies are decreased in agammaglobulinemia.

Immunoglobulin heavy chain deletion, a form of agammaglobulinemia, is a genetic disorder in which part of the antibody molecule is absent.

Severe combined immunodeficiency (SVID) is not precisely an immunoglobulin deficiency, but a combined deficiency resulting from a T-cell disorder.

Primary immunoglobulin deficiency syndromes occur only rarely.

Those that are X-linked occur more in males than females; other immunoglobulin deficiencies occur equally in both sexes.

Primary immunoglobulin deficiencies are primarily the result of congenital defects that affect the development and function of B lymphocytes (B cells), the white cells that fight infection and disease.

As they mature and develop memory, they switch to one of the other immunoglobulin classes.

Failure to switch or failure to make a subclass can lead to immunoglobulin deficiency diseases.

Immunoglobulin deficiency diseases cannot be cured, but treatment that replaces or boosts specific immunoglobulins can help support immune function in affected children.

The preferred treatment is to give specific immunoglobulins intravenously (immunoglobulin intravenous therapy or IVIG) or subcutaneously.

Parents with immunoglobulin deficient children and teenagers will likely be concerned that their children are in frequent contact with schoolmates and friends, the common route to infection.

Most allergies, including most drug allergies, occur because of a reaction with an immune system antibody called immunoglobulin E (IgE).

In CVID, immunoglobulin G (IgG) antibodies, one of several classes of antibodies, are either absent or produced in lower than normal numbers.

The toxin associated with tetanus, for example, is attacked by IgG1 and IgG3 antibodies; reduced percentages of either immunoglobulin subclass on a child's cells will leave the child unprotected against that specific toxin.

Serum immunoglobulin levels are measured in the clinical laboratory by a procedure called electrophoresis.

This is equivalent to 22 mg of the antibody immunoglobulin G (IgG) per kilogram of body weight.

The terms antibody and immunoglobulin are often used interchangeably, although immunoglobulin refers to the larger classification system for antibodies.

There are five types or classes of immunoglobulin that antibodies fit into, and each has a slightly different role in response against bacteria and viruses.

A type of B lymphocyte deficiency involves a group of disorders called selective immunoglobulin deficiency syndromes.

The most common type of immunoglobulin deficiency is selective IgA deficiency, occurring in about one in every 500 white persons.

In another immunoglobulin disorder, IgG and IgA antibodies are deficient, and there is increased IgM.

In this disorder, the production of one or more of the immunoglobulin types is decreased, and the antibody response to infections is impaired.

Patients with Bruton's agammaglobulinemia must be given periodic infusions of pooled immunoglobulin from multiple donors.

The product is called intravenous immunoglobulin (IVIG).

Over time, their immunoglobulin levels begin to decrease because they cannot successfully produce their own.

As the immunoglobulin levels decrease, the baby becomes increasingly vulnerable to bacterial infections.

Common symptoms of immunoglobulin deficiency usually appear after the infant is six months old.

After the first sting, the child's body produces an allergic substance called immunoglobulin E (IgE) antibody, which reacts with the insect venom.

Mast cells, found in the lining of the nasal passages and eyelids, display a special type of antibody called immunoglobulin type E (IgE) on their surfaces.

Mast cells, found in the lining of the nasal passages and eyelids, display a special type of antibody, called immunoglobulin type E (IgE), on their surface.

This immunoglobulin destroys any fetal blood cells in her bloodstream before her immune system can react to them.

Infants also start life with some immunoglobulin antibodies acquired from the mother.

Immunoglobulin antibodies are divided into five classes.

The capacity of the body to produce each immunoglobulin varies with age.

IgA or immunoglobulin A is a type of naturally-occurring antibody found in your respiratory and digestive tract.

An Fc receptor for human immunoglobulin G is located within the tegument of human cytomegalovirus.

In vivo immune evasion mediated by the herpes simplex virus type 1 immunoglobulin G Fc receptor.

A human T cell-specific cDNA clone encodes a protein having extensive homology to immunoglobulin chains.