Anemia Sentence Examples

He had pernicious anemia at age 49 years.

She had pernicious anemia, in which vitamin B12 is not absorbed from the intestine.

This is the very best way to confirm the presence of Feline Infectious Anemia.

Copper also helps us absorb iron and prevent anemia.

Autoimmune haemolytic anemia is a rare disease in which the body 's immune system seems to attack the red cells.

This form of MDS is called " refractory anemia with excess blasts " or RAEB.

Back to top Why might I be tested for sickle cell anemia?

If you have time, I have pernicious anemia and wondered if you had any information on.. .

Feline Infectious Anemia, a parasitic disease, is known by several names.

This creates severe anemia in the affected cat over time.

These cats have no signs or symptoms of illness and can live long lives as carriers of Feline Infectious Anemia.

All affected cats have anemia to varying degrees.

Reduced blood cell volume (hematocrit) is also considered anemia.

Little evidence of iron deficiency anemia was found from total iron deficiency anemia was found from total iron binding capacity.

People with Sickle Cell Anemia often have mild jaundice which can make the whites of their eyes look yellowish.

The patient displayed moderate leukocytosis, anemia and thrombocytopenia.

Pernicious anemia also causes soreness of the tongue, loss of weight, skin pallor often with a lemon tint, and intermittent diarrhea.

It also prevents pernicious anemia and is necessary to a healthy nervous system.

Autoimmune haemolytic anemia ABO and rh phenotype An ABO and full Rh phenotype are performed on all referrals.

In the clinic, the animals showing signs of anemia and lethargy have hematology and blood smears done virtually routinely.

Well diluted tincture in very small amounts has been used to treat anemia.

A slowly bleeding ulcer can also cause anemia, where there are not enough red blood cells to transport oxygen around the body.

But they may also have specific nutrient deficiencies such as iron deficiency anemia or low plasma zinc levels.

Strong need for adequate nutrition to prevent problems such as anemia and gestational diabetes.

A child may also develop extra vascular tissue with a deeper hemangioma which could cause platelet problems or anemia.

Since this disease can cause severe anemia, a transfusion may become necessary at some point.

When a cat is pregnant it is important to make sure that she does not have fleas that could cause anemia.

Fleas also feed on the feline's blood which can cause the animal to suffer anemia.

This is especially true if you're undernourished or have a health condition such as diabetes or anemia.

Don Quai (also called Anegelica sinensis, tang kui and female ginseng) is popular for treating gynecological conditions, mild anemia, fatigue and high blood pressure.

Long-term exposure to fleas can cause anemia, allergic dermatitis and intestinal parasites in your pet, so timely treatment is important.

Being underweight can cause anemia, dehydration, and hormonal problems.

Insufficient amounts of iron can lead to fatigue, memory loss, and anemia.

Deficiency vitamin B12 can lead to anemia and in severe cases, irreversible neurological damage.

As a result, anemia or nerve damage can be present without being diagnosed.

However, be aware that while folic acid can correct anemia, it cannot reverse nerve damage caused by a B12 deficiency.

A lack of B12 can lead to anemia, a blood disease that is characterized by a low level of red blood cells in the body.

One of the side effects of anemia is the slow breakdown of the nervous systems, which includes brain function and mood stabilization.

Vitamin B12 shots will increase the number and health of red blood cells, and treat the underlying anemia in the process.

Since red blood cells give you energy and stabilize nerve function, the symptoms of both anemia and depression will fade and eventually cease.

This is because anemia can slow down your metabolism due to lack of oxygen in the blood.

Recovering from anemia also causes an increase in energy, leading to more activity and exercise.

Vegans, who do not consume any animal products, can be at risk for a lack of vitamin B12 even if they don't suffer from pernicious anemia.

The preliminary report from the Los Angeles county coroner's office is revealing that Murphy died from complications from pneumonia, anemia and prescription drug toxicity.

As the parasites mature, they block the blood flow and can lead to anemia (reduced hemoglobin) or heart failure.

Yellow, blue, gray or white discoloration can indicate serious health problems such as internal bleeding, anemia or shock.

Historically, the herb has been used as a diuretic and as a remedy for certain illnesses, such as anemia.

If the disease is not treated, the parasitical infection can cause anemia, cardiac disease, kidney disease and endocrine failure.

Restless leg syndrome may be caused by anemia, pregnancy, some types of medications, alcohol, nicotine, or kidney disease.

Anemia - A severe iron deficiency can lead to cramping.

The most common causes of stroke are congenital (present at birth) and acquired heart diseases, and sickle cell anemia.

Epoetin aids the body in producing red blood cells and is currently used to treat anemia associated with kidney disease or caused by some drugs.

Although there is a high risk of repeat strokes in patients with sickle cell anemia, the risk can be reduced with regular blood transfusions.

Having a history of diseases that damage the bone marrow, such as aplastic anemia, or a history of cancers of the lymphatic system puts people at a high risk for developing acute leukemias.

The virus causes the destruction of red blood cells and, therefore, a deficiency in the oxygen-carrying capacity of the blood (anemia) can result.

In healthy children, the anemia is mild and only lasts a short while.

Symptoms of anemia include fatigue, lack of color, lack of energy, and shortness of breath.

In addition, there is a blood test for fifth disease, but it is generally used only for pregnant women and for people who have weakened immune systems or who suffer from blood disorders, such as sickle cell anemia.

There are tests and treatments, however, that can be performed on the fetus while still in the uterus that can reduce the risk of anemia or other complications.

Sickle cell anemia does not predispose newborn infants to jaundice.

Exchange transfusion corrects anemia associated with the destruction of red blood cells and is effective in removing sensitized red blood cells before they are destroyed.

With symptoms usually first appearing in childhood, the characteristics of this rare syndrome include pancreatic dysfunction and anemia (low red blood cells).

Sickle cell anemia, also called sickle cell disease (SS disease), is an inherited condition caused by having abnormal hemoglobin, the protein that carries oxygen in the blood.

Children with sickle cell anemia produce two abnormal hemoglobin proteins (inheriting one from each parent), which makes their red blood cells easily destructible while giving them a sickle-like shape.

Sickle cell anemia is usually inherited from parents who are carriers, who have the sickle cell trait-a milder form of sickle cell anemia, or one abnormal hemoglobin.

Sickle cell anemia and sickle cell trait are found mainly in people whose families come from Africa, the Caribbean, the Eastern Mediterranean, Middle East, and Asia.

In the United States, sickle cell anemia affects some 72,000 people.

Sickle cell anemia is caused by an error in the gene that signals the body how to make hemoglobin.

Children suffering from sickle anemia have episodes during which they suddenly become unwell or complain of severe abdominal or chest pain, headache, stiffness of the neck or drowsiness.

The diagnosis of sickle cell anemia is established during the newborn screen testing that is performed in the nursery at time of birth.

For children who are not tested, an electrophoresis test of the blood can detect the abnormal hemoglobin of sickle cell anemia.

In addition, the marrow must come from a healthy matched sibling donor and only about 18 percent of children with sickle cell anemia are likely to have a matched sibling.

Research contributed a great deal about sickle cell anemia from 1970 to the early 2000s concerning what causes it, how it affects the patient, and how to treat it.

Scientists were as of 2004 starting to be successful at developing drugs that prevent the symptoms of sickle cell anemia and procedures that they hope should eventually provide a cure.

It has been observed that some children with sickle cell anemia continue to produce large amounts of fetal hemoglobin after birth, and studies have shown that these children have less severe cases of the disease.

Children with sickle cell anemia are anemic to various degrees.

Most of the time they feel quite well, but if the anemia gets worse, they may feel very tired.

Sickle cell anemia is an inherited disease and lasts a lifetime.

Both sickle cell trait and sickle cell anemia are inherited.

If both parents have sickle cell trait and want to know whether the unborn child has sickle cell anemia, testing can be performed as early as the tenth week of pregnancy.

Parents should be aware that children with sickle cell anemia are also at increased risk of infection, especially from the Streptococcus pneumonia and H. influenzae bacteria.

Sickle cell anemia does not affect intelligence.

Sickle cell trait-Condition that occurs in people who have one of two possible genes responsible for the abnormal hemoglobin of sickle cell anemia.

People with this trait may suffer milder symptoms of sickle cell anemia or may have no symptoms.

Patients who have leukemia, polycythemia vera, or aplastic anemia are given periodic platelet count tests to monitor their health.

Leukemia and aplastic anemia can result in a low platelet count because of decreased production of platelets in the bone marrow.

Rarely, a Wilms' tumor is diagnosed after there has been bleeding into the tumor, resulting in sudden swelling of the abdomen and a low red blood cell count (anemia).

If the bone marrow is involved, anemia can result.

It causes an enlarged liver, bruising and skin lesions, anemia, enlarged lymph glands, other organ involvement, and extensive skull lesions.

This so-called "heterozygote advantage" is seen in some other genetic disorders, including sickle-cell anemia.

When CF is not treated for a longer period, a child may develop symptoms of malnutrition, including anemia, bloating, and, paradoxically, appetite loss.

Other symptoms include tiredness, pain, bone deterioration, broken bones, anemia, and increased bruising.

These can bleed and can, in turn, result in anemia.

An infection in pregnancy can cause the unborn baby to have severe anemia and the woman may have a miscarriage.

Late apnea can also affect full-term babies and may be a sign of an underlying problem such as congenital heart disease, infection, anemia, meningitis, or seizures.

A deficiency of folic acid may lead to anemia, in which there is decreased production of red blood cells.

Splenectomy can result in an increase and hemolytic anemia can result in a decrease in glycated hemoglobin.

Incompatibility between the Rh blood types of mother and child (mother Rh negative, baby Rh positive) can result in severe anemia in the baby (erythroblastosis fetalis).

The affected person may also require treatment such as intravenous fluids for complications of poisoning such as shock, anemia, and kidney failure.

Rarely, there are so many cysts that the kidneys functioning is impaired, or the cysts bleed, resulting in anemia.

Sometimes bed-wetting can be caused by a serious medical problem like diabetes, sickle-cell anemia, or epilepsy.

These include anemia, or breakdown of red blood cells; reduced platelets; reduced white cells; and bone marrow failure.

In some cases breath holding spells may be associated with anemia (a reduced number of red blood cells) caused by an iron deficiency, although this is controversial.

Anemia is a blood disorder characterized by abnormally low levels of healthy red blood cells (RBCs) or reduced hemoglobin (Hgb), the iron-bearing protein in red blood cells that delivers oxygen to tissues throughout the body.

Anemia can be mild, moderate, or severe enough to lead to life-threatening complications.

More than 400 different types of anemia have been identified.

Anemia in newborn infants is noted when hemoglobin levels are lower than expected for the birth weight and postnatal age.

Anemia can also be caused by the destruction of red blood cells or reduced red blood cell production.

Iron deficiency anemia is the most common form of anemia worldwide.

The onset of iron deficiency anemia is gradual and may not have early symptoms.

In this early stage of anemia, the red blood cells look normal, but they are reduced in number.

Symptoms of anemia, especially weakness and fatigue, develop at this stage.

Folic acid deficiency anemia is the most common type of megaloblastic anemia, arising from a problem with the synthesis of deoxyribonucleic acid (DNA) within the cells of the body.

Folic acid anemia is especially common in infants and teenagers.

Folic acid anemia can be a complication of pregnancy, when a woman's body needs eight times more folic acid than it does otherwise.

Less common in the United States than folic acid anemia, vitamin B12 deficiency anemia is another type of megaloblastic anemia that develops when the body does not absorb enough of this nutrient.

Pernicious anemia is the most common form of B12 deficiency.

Pernicious anemia is diagnosed more often in adults between ages 50 and 60 than in children or young people, although there is the possibility of inheriting the condition, with symptoms not appearing until later in life.

Anemia due to vitamin C deficiency is a rare disorder that causes the bone marrow to manufacture abnormally small red blood cells.

Hemolytic anemia can be present at birth (congenital hemolytic anemia or spherocytosis) or acquired later in life.

Hemolytic anemia can enlarge the spleen, an organ that also produces red blood cells when necessary.

Complications of hemolytic anemia in older children or adults include pain, gallstones, and other serious health problems.

Hemolytic disease of the newborn is a specific variation of hemolytic anemia in which an incompatibility exists between antigens on the cells of the mother and baby, causing antibodies to develop in the mother's circulation.

Hemolytic disease of the newborn and the anemia that results is detectable within the first few days after birth.

Depending on the strength of the antibody, the anemia may clear up on its own or exchange transfusions may be necessary to replace the newborn's blood.

An inherited form of hemolytic anemia, thalassemia comes from the production of abnormal hemoglobin.

Thalassemia major (homozygous thalassemia or Cooley's anemia) occurs in children in whom both parents pass on the genes responsible.

Sickle cell anemia is an inherited, chronic, incurable blood disorder that causes the body to produce defective hemoglobin, the abnormal HgbS, which occurs primarily in African Americans.

Cancer, chronic infection or inflammation, and kidney and liver disease often cause mild or moderate anemia.

Anemia can be the result of injuries, chronic or acute illnesses, complications of surgery or childbirth, metabolic disturbances or deficiencies, and adverse response to drug therapy administered for other conditions.

Malnutrition or malabsorption of nutrients can contribute to vitamin deficiency anemia and iron deficiency anemias.

Although red cell destruction and replacement is an ongoing process in the body, hereditary disorders and certain diseases can accelerate blood cell destruction, resulting in anemia.

However, excessive bleeding is the most common cause of severe anemia, and the speed with which blood loss occurs has a significant effect on the severity of symptoms.

Weakness, fatigue, and a run-down feeling may be the first signs of anemia.

Pasty or sallow skin color, or the absence of color in the gums, nail beds, creases of the palm, or lining of the eyelids are other signs of anemia.

In the United States, iron deficiency anemia is the most prevalent type of anemia, affecting about 240,000 toddlers between one and two years of age and 3.3 million women of childbearing age.

Anemia due to gradual blood loss is more common in women than in men, particularly pregnant women or women of menstruating age.

Pernicious anemia is more common in women and in African Americans and is less common in other racial groups.

Sickle cell anemia is more frequently diagnosed than thalassemias and occurs most often among African Americans.

When a child exhibits weakness, dizziness, listlessness, or fatigue, it may be the first sign of anemia.

Any prolonged bleeding or sudden blood loss requires examination by a physician and testing for anemia.

If anemia is due to chronic disease, there may be evidence of infection or inflammation.

Urine output may be reduced in severe anemia.

A reticulocyte (young RBCs) count will help determine if anemia is caused by impaired RBC production or increased RBC destruction.

Diagnosing thalassemia and sickle cell anemia, both of which involve disorders of hemoglobin, will require measuring the different types of hemoglobin through a laboratory testing method called hemoglobin electrophoresis.

In some anemias, a bone marrow sample will be removed (bone marrow biopsy) for microscopic examination, especially to confirm iron deficiency anemia or the megaloblastic anemias.

Anemia due to nutritional deficiencies can usually be treated with iron replacement therapy, specific vitamin supplements, or self-administered injections of vitamin B12.

People with folic acid anemia may be advised to take oral folic acid.

Vitamin B12 deficiency anemia requires a life-long regimen of B12 shots to maintain vitamin levels and control symptoms of pernicious anemia.

Anemia resulting from chronic disease is typically corrected by treating the underlying illness.

This type of anemia rarely becomes severe.

Sickle cell anemia will be monitored by regular eye examinations and diagnostic blood work.

Children with aplastic anemia are especially susceptible to infection.

Treatment for aplastic anemia may involve blood transfusions and bone marrow transplantation to replace malfunctioning cells with healthy ones.

Hemolytic anemia of the warm-antibody type may be treated with large doses of intravenous and oral corticocosteroids (cortisone).

Individuals who do not respond to medical therapy, may undergo surgery to remove the spleen, which controls the anemia in some individuals by helping to add more RBCs to the circulation.

There is no specific treatment for cold-anti-body hemolytic anemia.

Treatment of newborn anemia depends on the severity of symptoms, the level of Hgb, and the presence of any other diseases that may affect oxygen delivery, such as lung or heart disease or hyaline membrane disease.

The risk of transfusion (such as transfusion reactions, potential toxins, and infections such as HIV or hepatitis) are carefully weighed against the severity of the anemia in the infant.

Herbal supplements that will benefit individuals who have anemia include bilberry, dandelion, goldenseal, mullein, nettle, Oregon grape root, red raspberry, and yellow dock.

The diet is a ready source of nutrients that prevent and treat anemia.

The prognosis for anemias generally depends upon the severity of the anemia, the type of anemia, and the response to treatment.

The hereditary anemias, such as the thalassemias and sickle cell anemia, may require life-long treatment and monitoring whereas other types of anemia, once treated, are apt not to recur.

Severe anemia may lead to other serious conditions, particularly if oxygen delivery is compromised for long periods of time or RBC destruction is more rapid than can be controlled by normal RBC replacement or specific treatment.

Severe blood loss or prolonged anemia can result in life-threatening complications.

A wholesome, balanced diet rich in nutrients can help prevent dietary deficiencies that lead to anemia.

Regular physical examinations can help evaluate a child's overall health and reveal possible signs or symptoms of anemia.

Everything You Need to Know about Anemia.

A low red blood cell count can lead to anemia (deficiency of red blood cells) and fatigue.

Low blood cell counts caused by the effect of chemotherapy on the bone marrow can lead to anemia, infections, and easy bleeding and bruising.

Patients with anemia have too few red blood cells to deliver oxygen and nutrients to the body's tissues.

These conditions cause varying degrees of anemia, which can range from insignificant to fatal.

Alpha thalassemia major, sometimes called hemoglobin Barts or hydrops fetalis, is a fatal disease that results in severe anemia that begins even before birth.

Beta thalassemia, also called Cooley's anemia, is the most well known type of thalassemia.

Beta thalassemia causes variable anemia that can range from moderate to severe, depending in part on the exact genetic change underlying the disease.

Beta thalassemia major causes severe anemia that usually occurs within three to six months after birth.

If left untreated, severe anemia can result in stunted growth and development, as well as other characteristic physical complications that can lead to a dramatically decreased life expectancy.

Beta thalassemia intermedia is a clinical term that describes the disease in individuals who have moderate anemia that only requires blood transfusions intermittently.

Carrying the trait is generally thought not to cause health problems, although some women with beta thalassemia trait may have an increased tendency toward anemia during pregnancy.

It is important to be able to recognize the signs of severe anemia that require medical attention.

It is also important to be aware of the medications, chemicals, and other exposures to avoid due to the theoretical risk they pose of precipitating a severe anemia event.

When severe anemia occurs, it is treated with blood transfusion therapy.

Affected fetuses develop severe anemia as early as the first trimester of pregnancy.

Beta thalassemia major is characterized by severe anemia that can begin several months after birth.

Severe anemia taxes other organs in the body such as the heart, spleen, and liver, which must work harder than usual.

This factor allows for treatment with blood transfusion therapy, which can prevent most of the complications of the severe anemia caused by beta thalassemia major.

Individuals with beta thalassemia intermedia have a more moderate anemia that may only require treatment with transfusion intermittently, such as when infections stress the body.

This helps prevent severe anemia and allow for growth and development that is more normal.

Common underlying disorders include leukemia, drug toxicity, or aplastic anemia, all of which lead to decreased or defective production of platelets in the bone marrow.

Knowing this level may help doctors evaluate low concentrations of normal hemoglobin in red blood cells (anemia), as well as higher-than-normal levels of fetal hemoglobin or its hereditary persistence.

Fetal hemoglobin measurement helps diagnose a group of inherited disorders that affect hemoglobin production, among which are the thalassemias and sickle cell anemia.

It may also be done to help doctors diagnose acquired illnesses such as acquired hemolytic anemia, leukemia, pernicious anemia, and certain types of cancer.

For example, HbF can be found in higher levels in sickle cell anemia and other hereditary anemias.

It may also reappear in adults when the bone marrow is overactive, as in disorders such as pernicious anemia, multiple myeloma, and invasive (metastatic) cancer affecting bone marrow.

Sickle cell anemia, the inherited condition characterized by curved (sickle-shaped) red blood cells and chronic hemolytic anemia, is an example of the first category.

Hemolytic anemia-A form of anemia characterized by chronic premature destruction of red cells in the bloodstream.

Women at risk for asphyxia neonatorum pregnancies should receive focused prenatal care from an obstetrician skilled at preventing and detecting problems such as anemia that may contribute to asphyxia neonatorum.

For example, doctors may order a test of iron levels in the blood because low levels of iron (anemia) may accompany celiac disease.

Secondary complications, such as anemia and osteoporosis, resolve in almost all patients.

By 1997, approximately 800 different diagnostic tests were available, most of them for hereditary genetic disorders such as Tay-Sachs disease, sickle cell anemia, hemophilia, muscular dystrophy, and cystic fibrosis.

Folate deficiency causes megaloblastic anemia, which is characterized by the presence of large abnormal cells called megaloblasts in the circulating blood.

The symptoms of megaloblastic anemia are tiredness and weakness.

Vitamin B12 deficiency causes megaloblastic anemia and, if severe enough, can result in irreversible nerve damage.

Pernicious anemia, homocystinuria, and biotinidase deficiency are three examples of genetic diseases which are treated with megadoses of vitamins.

One example is pernicious anemia, a disease that tends to occur in middle age or old age and impairs the absorption of vitamin B12.

If left untreated, pernicious anemia leads to nervous system damage.

Young women within the first menstrual period are not usually treated unless symptoms are exceptionally severe or if anemia develops.

Blood tests show a high white blood cell count, high platelet count, a high level of protein in the blood serum, and mild anemia.

Infants, people with heart or lung disease, or those with anemia may be more seriously affected.

Autoimmune diseases such as autoimmune hemolytic anemia (AIHA), immune thrombocytopenia purpura (ITP), rheumatoid arthritis, autoimmune thyroiditis, and systemic lupus erythematosus are sometimes associated with CVID.

A blood test for anemia may be performed at this visit if it was not done at the nine-month exam.

About 10 percent of these babies will be born with obvious problems, including prematurity, lung problems, an enlarged liver and spleen, jaundice, anemia, low birth weight, small head size, and inflammation of the retina.

The condition may also be associated with protein-losing enteropathy, low levels of iron in the blood serum or in the bone marrow (iron-deficiency anemia), or impaired absorption of nutrients by the intestines (malabsorption).

Protein-losing enteropathy may lead to abnormally large amounts of fluid in the intercellular tissue spaces of the body (edema), abdominal distension, and lack of red blood cells (anemia).

Blood loss is usually minimal, and anemia is rare.

Iron deficiency anemia refers to anemia that is caused by lower than normal levels of iron.

This type of anemia is caused by deficient erythropoiesis, the ongoing process of the bone marrow to produce healthy red blood cells (RBCs).

Anemia resulting from a deficiency of iron is also called microcytic anemia.

Anemia is a blood disorder characterized by abnormally low levels of healthy RBCs or reduced levels of hemoglobin (Hgb), the iron-bearing protein in RBCs that delivers oxygen to tissues throughout the body.

Blood cell volume (hematocrit) may also be reduced in some anemias, but not necessarily in iron deficiency anemia.

In developing countries in tropical climates, the most common cause of iron deficiency anemia is infestation with hookworm.

It is accepted that iron is hard to absorb; this, in combination with diets that may not meet daily requirements, is a common route to iron deficiency and iron deficiency anemia.

Blood losses from gastrointestinal bleeding, excessive menstrual bleeding, and infection with hookworm can deplete iron and lead to iron deficiency anemia.

The symptoms of iron deficiency anemia appear slowly and typically include weakness and fatigue.

Weakness, dizziness, listlessness, or fatigue may be the first signs of iron deficiency anemia.

In the United States, iron deficiency anemia affects thousands of toddlers between one and two years of age and more than 3 million women of childbearing age.

Diagnosing iron deficiency anemia begins with the pediatrician taking a careful history, including the child's age, symptoms, illnesses, general state of health, and a family history of anemias.

In iron deficiency anemia, the RBC count can be normal or elevated and hemoglobin will be abnormally low.

In infants, iron deficiency anemia is defined as having a hemoglobin level below 109 mg/ml when measured in whole blood, and a hematocrit of less than 33 percent.

A reticulocyte (young RBCs) count will help determine if anemia is caused by impaired RBC production, as in iron deficiency anemia, or increased RBC destruction as in some other types of anemia.

Protoporphyrin IX, a component of hemoglobin, may be measured to help confirm a diagnosis of iron deficiency anemia.

In this case, a diagnosis of iron deficiency anemia may include examination with a sigmoidoscope, a flexible, tube-like instrument with a light source that permits examination of the colon.

The diagnosis of iron deficiency anemia may include a test for oral iron absorption, especially when evidence suggests that oral iron supplements have failed to raise hemoglobin.

The goal of treatment for iron deficiency anemia is to restore iron levels and the production of healthy RBCs and increase the essential flow of oxygen to tissues.

Treatment of iron deficiency anemia sometimes requires more than iron supplementation.

When iron deficiency is provoked by hemorrhoids or gastrointestinal bleeding, for example, surgery may be required to prevent recurrent iron deficiency anemia.

Herbal supplements that benefit individuals who have iron deficiency anemia include alfalfa, burdock root, dandelion, dong quai, mullein, nettle, raspberry leaf, shepherd's purse, and yellow dock.

Decreased dietary iron intake is a contributing factor in iron deficiency and iron deficiency anemia.

The prognosis for treating and curing iron deficiency anemia is excellent, particularly when those affected take iron supplements as advised and are able to assimilate the iron.

A number of studies have shown that iron deficiency anemia in infancy can result in reduced intelligence, when intelligence was measured in early childhood.

It is not certain if iron supplementation of children with reduced intelligence, due to iron-deficiency anemia in infancy, has any influence in allowing a "catch-up" in intellectual development.

Iron deficiency anemia in infants and young children can be prevented by breast-feeding, consuming good dietary sources of iron, and using fortified foods.

Understanding iron metabolism and the ways to ensure that iron deficiency anemia in infants and children can be successfully treated and prevented from recurring may be concerns of parents.

Most people with this disorder have frequent infections, and some also experience auto-immune phenomena, such as autoimmune hemolytic anemia or rheumatoid arthritis.

Other laboratory findings that are associated with hemophilus infections include anemia (low red blood cell count) and a drop in the number of white blood cells in children with severe infections.

Iron deficiency causes anemia (low hemoglobin and reduced numbers of red blood cells), which results in tiredness and shortness of breath because of poor oxygen delivery.

Signs of copper deficiency may include anemia, diarrhea, weakness, poor respiratory function, baldness, skin sores, and increased lipid (fat) levels in the blood.

Symptoms of iron deficiency include anemia and resulting fatigue and weakness, especially during physical exertion.

Anemia and other effects of iron deficiency are not usually life-threatening and can be corrected with supplementation.

Anemia, diarrhea, and fluid and electrolyte disorders are common.

Red blood cell (RBC) indices are calculations derived from the complete blood count that aid in the diagnosis and classification of anemia.

Red blood cell indices help classify types of anemia, a decrease in the oxygen carrying capacity of the blood.

Anemia is diagnosed when either the hemoglobin or hematocrit of a blood sample is too low.

The mechanisms by which anemia occurs will alter the RBC indices in a predictable manner.

Therefore, the RBC indices permit the physician to narrow down the possible causes of an anemia.

This occurs in many diseases, including iron deficiency anemia, thalassemia (an inherited disease in which globin chain production is deficient), and anemias associated with chronic infection or disease.

The most common causes of macrocytic anemia are vitamin B 12 deficiency, folate deficiency, and liver disease.

Normocytic anemia may be caused by decreased production (e.g. malignancy and other causes of bone marrow failure), increased destruction (hemolytic anemia), or blood loss.

Iron deficiency is the most common cause of a hypochromic anemia.

For example, thalassemia minor and iron deficiency anemia are both microcytic and hypochromic anemias, and overlap in MCV and MCH.

However, iron deficiency anemia has an abnormally wide RDW, but thalassemia minor does not.

When the hematocrit is determined by centrifugation, the MCV and MCHC may differ from those derived by an electronic cell counter, especially in anemia.

The indices are used to help in the differential diagnosis of anemia.

When fibrous or bony tissue invades bone marrow where red blood cells are made, the individual may develop anemia.

The severity of anemia seems to determine the course of an individual's osteopetrosis.

The hemangiomas are large and may increase in size rapidly and may cause severe anemia in infants.

Some patients with polyarticular JA will have other symptoms of a systemic illness, including anemia (low red blood cell count), decreased growth rate, low appetite, low-grade fever, and a slight rash.

As with any chronic disease, anemia may be noted.

Severe diseases in the liver or kidneys, certain infections, sickle cell anemia, and some cancers also affect gonads.

Other tests may include a thyroid level; sperm count; prolactin level (milk hormone); blood tests for anemia, chemistries, and iron; and genetic analysis.

Some patients also have too few red blood cells (anemia) and an enlarged spleen (splenomegaly).

The resulting anemia may be so profound that the fetus may die in utero.

Reacting to the anemia, the fetal bone marrow may release immature RBCs, or erythroblasts, into the fetal peripheral circulation, causing erythroblastosis fetalis.

The antibodies cause the baby's red blood cells to be destroyed and the baby develops anemia.

The baby's body tries to compensate for the anemia by releasing immature red blood cells, called erythroblasts, from the bone marrow.

Hydrops fetalis and anemia can also contribute to heart problems.

In cases in which incompatibility is not identified before birth, the baby suffers recognizable characteristic symptoms such as anemia, hyperbilirubinemia, and hydrops fetalis.

Other blood tests reveal anemia, abnormal blood counts, and high levels of bilirubin.

One or more transfusions may be necessary to treat anemia, hyperbilirubinemia, and bleeding.

When people with this condition take certain drugs, their red blood cells break down, causing anemia.

They should also be used with extreme care in patients with liver problems, kidney problems, and some types of anemia.

Anemia, malnutrition, and digestive disorders, including bowel obstructions, can develop, if trichotillomania develops into trichotillophagia or eating of the hairs.

Some pharmaceutical companies claim that anemia causes hair loss.

Anemia is a decrease in red blood cells, often related to iron deficiency.

Lack of it may lead to anemia, which in turn can cause loss and shedding.

Both partners should have a medical consultation to discover and treat any problems, such as iron-deficiency anemia.

Pregnant teens are more likely to experience anemia and preeclampsia than older mothers.

Anemia. Being anemic means you have a shortage of red blood cells, often due to not getting enough iron in your diet.

Anemia can make you feel run-down and tired.

You doctor or midwife may need to check your kidney or liver function, re-check you for anemia, or look for other problems.

When iron levels are low, anemia can result.

Teen moms are at higher risk than older women for pregnancy problems like high blood pressure and anemia.

Because lupus antibodies can cross the placenta to the fetus, the baby may experience a low white blood cell count, anemia, or a slow heartbeat.

During this stage, doctors will monitor babies closely for signs of jaundice, anemia, and infections.

This can be a significant concern because heavy bleeding can lead to anemia; you should let your doctor know about this problem so that he/she can monitor your blood count.

You may also need to take iron supplements during this heavy bleeding to prevent anemia.

WebMD talks about severe anemia as it relates to B12 deficiency.

A deficiency of vitamin B12 can result in anemia and result in a far reaching effect on the overall health of the body.

The most significant side effect of a vitamin B12 deficiency is pernicious anemia and this may be accompanied by neurological problems.

People who suffer from pernicious anemia were historically advised to eat plentiful amounts of liver.

It helps prevent anemia and works with folic acid to regulate red blood cells.

People who abuse alcohol or those with anemia may need a supplement in case of a folic acid deficiency.

Blood tests can detect vitamin and mineral deficiencies such as anemia (iron deficiency), B12 and vitamin D deficiencies.

People taking iron supplements to treat iron-deficiency anemia are also advised to take ascorbic acid or eat a food containing vitamin C when they take their iron.

Taking iron with vitamin C can help to more effectively prevent and treat anemia.

It is essential to many processes, including prevention of anemia during pregnancy.

Deficiency signs and symptoms include anemia, sore tongue, loss of appetite, weakness, failure to grow or slow growth in children, and digestive upset.

It is essential in red blood cell formation, and folic acid deficiency can lead to macrocytic anemia.

Blood donations benefit premature babies, cancer patients, individuals with sickle cell anemia and individuals undergoing surgery.

If the person continues to consume gluten, without any type of treatment, he or she could face serious complications, including iron deficiency anemia, osteoporosis and even some forms of lymphoma.

Are the medications you take, such as prescriptions for ADD or ADHD, fibromyalgia, osteoporosis, anemia, autism, or other illnesses needed as the result of gluten intolerance?

Over time, this can lead to conditions such as anemia, osteoporosis, and tooth decay.

The lack of nutrients makes you vulnerable to other conditions such as loss of bone density and anemia.

As a result of the damage to the small intestine and villi, poor absorption can lead to iron deficiencies and anemia.

In children with weakened immune systems or with anemia, the virus can be quite serious causing severe anemia.

Joint pain, muscle cramps, anemia, mouth sores, skin rash, dental disorders and tingling in the legs and feet are sometimes present as symptoms of gluten intolerance.

This can help to avoid potential complications associated with osteoporosis, certain types of cancer or anemia.

As things deteriorate, anemia and color blindness can set in, as does a higher risk of cardiovascular ailments.

They are often associated with a host of serious conditions, including anemia, malnutrition, respiratory disease, renal failure and kidney problems.