The choline found in lecithin is used to make acetylcholine in the body.
Drug A attaches to the acetylcholine receptors on the smooth muscle without stimulating them and prevents acetylcholine from gaining access to them.
Also, choline is used to produce the important neurotransmitter acetylcholine.
They always use the neurotransmitter acetylcholine, and are always excitatory.
acetylcholine in the brain.
However, Reminyl also appears to act on the nicotinic neuronal receptors in the body, making them release more acetylcholine.
When enough acetylcholine attaches itself to the outer surface of a muscle cell, the muscle cell becomes ' excited enough ' to contract.
acetylcholine receptor proteins in the brain, which are made up of several different subunits.
acetylcholine release at the nerve terminal.
acetylcholine levels in the brain may affect mood disorders, and supplemental choline can increase acetylcholine levels.
acetylcholine receptor antibody.
acetylcholine esterase inhibitors.
acetylcholine receptor gene family of Drosophila melanogaster.
Many types of smooth muscle also contain gap junctions and muscarinic acetylcholine receptors, but here acetylcholine normally leads to contraction.
Alzheimer's disease is caused by the destruction of certain brain cells leading to the loss of the neurotransmitter acetylcholine.
The Cambridge University research, published in the journal Brain, suggests boosting the chemical acetylcholine may limit the effect of this damage.
Choline is transformed into the brain neurotransmitter acetylcholine, which is vital to the efficient transmission of brain signals.
Terminal nerve fibers release acetylcholine into the neuromuscular junction, which then binds to acetylcholine receptors on muscle cells.
antagonist at acetylcholine receptors which dilated their pupils.
The first medicines assessed for Alzheimer's treatment work in this way and are called acetylcholine esterase inhibitors.
We are currently focussed on two distinct families of these receptors: 1. The ionotropic glutamate receptors and 2. The nicotinic acetylcholine receptor.
There was no significant difference between groups in MMH or in the response to acetylcholine iontophoresis.
These may come from new approaches such as the role of sigma, NMDA and acetylcholine receptors.
vasodilator effects of acetylcholine in humans have appeared in prestigious journals from 1987 until the present.
Papers describing the coronary vasodilator effects of acetylcholine in humans have appeared in prestigious journals from 1987 until the present.
Specifically, acetylcholine, a chemical in the body that transmits nerve signals, is released from nerve endings and stimulates secretion of sweat.
Anticholinergic drug-Drugs that block the action of the neurotransmitter acetylcholine.
"The Importance of Nicotinic Acetylcholine Receptors in Schizophrenia, Bipolar Disorder, and Tourette's Syndrome."
Also, botulinum toxin is a neurotoxin that blocks the ability of motor nerves to release acetylcholine, the neurotransmitter that relays nerve signals to muscles.
These proteins control the release of acetylcholine, a neurotransmitter that stimulates muscle cells.
With acetylcholine release blocked, nerves are not able to stimulate muscles.
Anticholinergics, including trihexyphenidyl (Artane) and benztropine (Benztrop MES, Cogentin), block acetylcholine receptors in the brain.
Acetylcholine receptors are integral proteins that respond to the neurotransmitter acetylcholine by opening a pathway in the membrane for ion diffusion across the cell membrane.
The drug boosts the function of a key brain chemical called acetylcholine.
The junction shown in figure 1 is of a nerve that releases acetylcholine.
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