Atrioventricular canal defect (also called endocardial cushion defect or atrioventricular septal defect) is a large hole in the septum, accompanied by abnormal tricuspid and mitral valves that are not formed as individual valves.
Pulmonary artery banding, narrowing the pulmonary artery with a band to reduce blood flow and pressure in the lungs, is used for ventricular septal defect, atrioventricular canal defect, and tricuspid atresia.
Cyanotic defects include truncus arteriosus, total anomalous pulmonary venous return, tetralogy of Fallot, transposition of the great arteries, tricuspid atresia, and pulmonary atresia.
For tetralogy of Fallot, tricuspid atresia, or pulmonary atresia, the shunt procedure creates a passage between blood vessels, sending blood into parts of the body that need it.
For tricuspid atresia and pulmonary atresia, the Fontan procedure connects the right atrium to the pulmonary artery directly or with a conduit, and the atrial defect is closed.
Ebstein's anomaly is a rare congenital syndrome that causes malformed tricuspid valve leaflets, which allow blood to leak between the right ventricle and the right atrium.
In tricuspid atresia, the baby lacks a triscupid valve and blood cannot flow properly from the right atrium to the right ventricle.
Treatment often involves repairing the tricuspid valve.
There are but three pairs of incisor teeth in each jaw, and the upper molars are tricuspid.
The communication with the atrium is guarded by a valvula cardiaca dextra, which only in function represents the mammalian tricuspid; it consists of an oblique reduplication of the muscular fibres together with the endocardiac lining of the right ventricle, while the opposite wall is convex and forms neither a velum nor papillary muscles, nor chordae tendineae.